Low Birthweight Piglets Essay -- Food, Pork Industry

entreLow fork up weight piglets have high mortality and short(p) emergence postnatally. The pork industry has strategies to increase the piglets birth weight. Maternal growth hormone sermon with developmental timing and dose difference change magnitude foetal growth in pigs 1-4. As GH cannot cross the placenta 5, the increase in fetal growth must due to changes in parental(p) metabolism and/or placental development and function. Placental weight was increased with maternal GH treatment 4. However, none of the above studies has demonstrated the effect of maternal GH treatment on placental structural development and function in pigs therefore, this is the pass that will be addressed in this study. Birth WeightDe considerationinants of fetal growth and birth weight in pigsBirth weight is influenced by several factors during pregnancy, such as parity, maternal nutrition, uterine capacity and pack size. Pigs have two parity groups, sows and gilts. Sows are pigs which have given birth at least 3 times before, and gilts, are pigs that had never been pregnant. The outset birth weight of the progeny from gilts might be due to startle pregnancy. As gilts are growing when they pregnant, so mother and foetus were competing for limit nitrogenous substrates to meet their needs, and leads to low birth weight in fetus *gatford 2009 & Schoknecht 1993.Piglets from dam which has restriction in food intake or particularised nutrients during gestation have reduced in birth weight. The progeny of gilts ply with protein-deficient diet (0.5% protein) in early (d 1 to d44) or late (d 82 to term) during gestation have demoralize birth weight compared to the control (with 13% protein in diet) whereas protein deficient diet throughout pregnancy caused the progeny weigh... ...reased maternal tippytoe meat percentage *rehfeldt 2001. The backfat depth of pGH treated dams in *gatford 2010 was lower than the controls at farrowing and weaning *. This suggested that GH treatme nt can stimulate lean growth and inhibiting adipose tissue growth in pigs. The gestation length of sows, but not gilts was being shortened by long term GH treatment.Maternal GH treatment would also affect the concentration of maternal circulate metabolites and hormones. There was an increased in amino acids nitrogen and decreased in free fatty acids in maternal circulation by a 2 or 4 mg GH dose treatments from d 25 to d 51(gatford, 2000). Maternal plasma urea concentration was decreased by 28% by GH treatment with a dose 15ug/kg from days 25 to 50 *gatford 2009. There was a similar finding in other study with maternal GH treatment in underfed gilts *gatford 2000.